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Possible through diffused modulatory inputs that regulate dendritic plasticity Magee and Johnston 2005 ; . We should note, however, that our observed gain change is opposite in sign to that recently proposed by Larkum and colleagues 2004 ; , raising the possibility of multiple gain control mechanisms in dendrites of different classes of neurons. That changing the variance of the input produced a gain change with no change in the filter properties of the input output function is somewhat surprising given that our highvariance input was large enough to produce action potentials. This is because mechanisms that could potentially modify gain, such as the opening of dendritic voltage-gated conductances, would also be expected to change the temporal filtering properties of the dendrite. Neuronal mechanisms that provide multiplicative changes in gain with no change in the temporal properties have not been easy to resolve and represent an active area of neuroscience investigation Salinas and Thier 2000 ; . Functional implications of the LN model. Table of Contents We expect to continue to devote substantial resources to research and development in future periods as we continue our current clinical trials and start additional trials. Research and development expenses will likely increase on an annual basis but may fluctuate from period to period based largely on clinical trial activities. These costs could even decrease from quarter to quarter as we complete enrollment and patient treatment in current clinical trials. General and Administrative General and administrative expenses were .2 million and .6 million for the years ended December 31, 2005 and 2004, respectively. The .6 million increase in general and administrative expenses primarily reflects additional expenses associated with becoming a public company in 2005, including .2 million of higher legal fees, insurance premiums, and consulting services; and .2 million of expenses related to higher staffing levels. Additionally, the increase in general and administrative expenses in 2005 compared to 2004 was due to ##TEXT##.7 million of increased patent expenses and a ##TEXT##.5 million increase in non-cash stock-based compensation expenses. We expect our general and administrative expenses to continue to increase due to additional administrative and infrastructure costs as well as costs associated with implementing procedures for compliance with Section 404 of the Sarbanes-Oxley Act of 2002. Interest and Other Income Interest income for the year ended December 31, 2005 was .2 million compared to ##TEXT##.4 million for the year ended December 31, 2004. The increase was due to greater invested cash balances due to proceeds received from our initial public offering completed in February 2005 and our follow-on offering completed in October 2005, as well as higher average interest rates in 2005. Interest Expense Interest expense for the years ended December 31, 2005 and 2004 was , 000 and , 000, respectively, reflecting the declining balance of our note payable. Results of Operations for the Years Ended December 31, 2004 and 2003 Research and Development Research and development expenses for the year ended December 31, 2004 were .3 million compared to .3 million for the year ended December 31, 2003. The .0 million increase in research and development expenses in 2004 was due primarily to increases of .5 million for clinical trial costs, .9 million for increased staffing, .5 million for licensing costs, ##TEXT##.9 million for clinical drug supply, ##TEXT##.3 million for facility and related costs and .6 million for non-cash stock-based compensation. Research and development expenses associated with glufosfamide were .5 million for the year ended December 31, 2004 and ##TEXT##.1 million for the year ended December 31, 2003. This increase was due to the activities leading up to and initiation in 2004 of a Phase 3 clinical trial for the second-line treatment of pancreatic cancer. Research and development expenses associated with TH-070 increased to .3 million for the year ended December 31, 2004 from ##TEXT##.4 million for the year ended December 31, 2003, due to the Phase 2 trial conducted in 2004. Research and development expenses associated with 2DG were .8 million for the year ended December 31, 2004, and .2 million for the year ended December 31, 2003. This decrease is a result of the completion of a major portion of preclinical studies during 2003. Discovery research expenses were .7 million for the year ended December 31, 2004 and .6 million for the year ended December 31, 2003. The increase in discovery research expenses was primarily due to increased staffing. 49. At 1 You can see here, what pediatricians 3 4 5 corporis surprised associated don't to stay turns away from out they are using. the high-potency more Lotrisone 2 steroid at least drug. for corticosteroid You can see they topical than monotherapy, really It and buy nizoral.Buy lotrisone lotion
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